Recent years have witnessed considerable growth in the field of psychoneuroimmunology, which describes how psychological factors, such as stress and depression, impact the neurological and immune systems. Research increasingly indicates that psychological states play a key role in the development and exacerbation of inflammatory diseases such as heart disease, diabetes, multiple sclerosis, Alzheimer's, and autoimmune disorders. In this book, editor Kathleen Kendall-Tackett and an elite group of researchers explore the ways physical and psychological stressors such as poor sleep, PTSD, and depression, trigger the inflammatory response and increase the risk of disease. They approach this material from a variety of perspectives. Chapters in Part I describe the biological processes involved in inflammation, focusing on both the typical bodily response to threat as well as on the long-term deleterious effects of stress upon the immune system; while chapters in Part II examine the role of psychosocial stress in disease etiology. Throughout, chapter authors present evidence of connections between mind and body, and emphasize the need for improved communication between physicians and mental health care providers. This book will be a valuable resource for researchers as well as practitioners who hope to share the benefits of these findings with their clients.

Kathleen A. Kendall-Tackett; American Psychological Association

edited by Kathleen Kendall-Tackett

American Psychological Association (APA)

American Psychological Association, Washington, DC, 2010

United States, United States of America

Illustrated, 1, PS, 2009

Includes bibliographical references and index.

"Why are depressed people more likely to suffer heart attacks? Why do people with hostile worldviews have higher rates of metabolic syndrome and Type 2 diabetes? Recent years have witnessed considerable growth in the field of psychoneuroimmunology, which describes how psychological factors, such as stress and depression, impact the neurological and immune systems. Research increasingly indicates that psychological states play a key role in the development and exacerbation of inflammatory diseases such as heart disease, diabetes, multiple sclerosis, Alzheimer's, and autoimmune disorders. In this book, Kathleen Kendall-Tackett and an elite group of researchers explore the ways physical and psychological stressors such as posttraumatic stress disorder, depression, and poor sleep trigger the inflammatory response and increase the risk of disease. They attack these issues in a variety of ways: Chapters in Part I describe the biological processes involved in inflammation, focusing on both the typical bodily response to threat as well as on the long-term deleterious effects of stress upon the immune system, and chapters in Part I I examine the role of psychosocial stress in disease etiology. Throughout, chapter authors present evidence of connections between mind and body and emphasize the need for improved communication between physicians and mental health care providers. This book will be a valuable resource for researchers as well as practitioners who hope to share the benefits of these findings with their clients"--Jaquette. (PsycINFO Database Record (c) 2010 APA, all rights reserved)

"Why are depressed people more likely to suffer heart attacks? Why do people with hostile worldviews have higher rates of metabolic syndrome and Type 2 diabetes? Recent years have witnessed considerable growth in the field of psychoneuroimmunology, which describes how psychological factors, such as stress and depression, impact the neurological and immune systems. Research increasingly indicates that psychological states play a key role in the development and exacerbation of inflammatory diseases such as heart disease, diabetes, multiple sclerosis, Alzheimer's, and autoimmune disorders. In this book, Kathleen Kendall-Tackett and an elite group of researchers explore the ways physical and psychological stressors such as posttraumatic stress disorder, depression, and poor sleep trigger the inflammatory response and increase the risk of disease. They attack these issues in a variety of ways: Chapters in Part I describe the biological processes involved in inflammation, focusing on both the typical bodily response to threat as well as on the long-term deleterious effects of stress upon the immune system, and chapters in Part I I examine the role of psychosocial stress in disease etiology. Throughout, chapter authors present evidence of connections between mind and body and emphasize the need for improved communication between physicians and mental health care providers. This book will be a valuable resource for researchers as well as practitioners who hope to share the benefits of these findings with their clients"--Jacket. (PsycINFO Database Record (c) 2010 APA, all rights reserved)

An overview of stress, immunity, and risk of disease / Maureen Groër, Mary Meagher & Kathleen Kendall-Tackett
Inflammation, fatty acid oxidation, and neurodegenerative disease / Mary Nivison, Angela L. Guillozet-Bongaarts, and Thomas J. Montine
Sleep and inflammation : a potential link to chronic diseases / Edward C. Suarez and Harold Goforth
Polyunsaturated fatty acids, inflammation, and inflammatory diseases / Philip C. Calder
Depression, hostility, posttraumatic stress disorder, and inflammation : the corrosive health effects of negative mental states / Kathleen A. Kendall-Tackett
Cognitive and behavioral reactions to stress among adults with PTSD : implications for immunity and health / Jeffrey L. Kibler, Kavita Joshi, and Erin E. Hughes
Social stress and inflammation in the exacerbation of multiple sclerosis : an animal model with implications for humans / Mary W. Meagher and C. Jane R. Welsh
Allostasis : a model for women's health / Maureen Groër and the Women's Health Group
Treatments for depression that lower inflammation : additional support for an inflammatory etiology of depression / Kathleen Kendall-Tackett
Inflammation and chronic disease : clinical implications and future directions / Kathleen Kendall-Tackett.